ANSWERS TO IN-CHAPTER QUESTIONS

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GUIDANCE |
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Q1.
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The
blind spot on the Retina has no rod or cone cells; so no (image) impulse is
transmitted to the brain. Each eye
has a different area of vision missing.
The fusion of messages from both eyes enables the brain to fill in the
“gap” in the picture. |
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Q2.
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This
corrects the deformation of the capsule that is pressing on the sensitive
nerve endings at the centre of the pacinian corpuscle. The stretched nerve membrane now returns
to its original shape shutting the gates of the sodium channel proteins. The nerve membrane goes into the
refractory period since an action potential was previously generated. This enables the person to feel the
pressure has been released. |
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Q3.
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Air
® cornea Cornea
® aqueous humour Aqueous
humour ® lens Lens
® vitreous
humour |
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Q4.
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Excitatory. |
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Q5.
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The
light is more likely to impinge on the rods of the area around fovea. You can see more stars on a dark night out
of the “corners” of your eyes. In dim
light only rods are activated, so colour vision is lost as well as acuity. |
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Q6.
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Poor
vision in dim light conditions; insufficient retinal to combine with opsin to
form rhodopsin which is needed for the rod cells to work. |
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Q7.
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a) Actin in the protein present in actin filaments,
which are much thinner than myosin filaments. Therefore actin filaments cause lighter striations. Actin filaments are attached to plates
called Z lines. The myosin and actin
filaments slide over each other so that their overlap increases during muscle
contraction. b)
A = A Band |
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Q8.
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The
A Band stays the same. The I Band all
but disappears, i.e. it narrows, as the filaments slide one another, i.e.
they interdigitate. Interlocking
figners is a good analogy – pushing them together shortens the distance they
span. |
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Q9.
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The
actin potential spreads down the membranes of the T-tubules. This causes the membranes of the nearby
sarcoplasmic reticulum to become more permeable and Ca2+ ions
diffuse out rapidly. The Ca2+
ions reach the actin filaments in the myofibrils, unblocking the binding
sites and causing contraction, i.e. actomyosin cross bridges form. The Ca2+ ions attach to
troponin, which causes the tropomyosin to change position, unblocking the
binding site, i.e. the cross bridges can form. No energy is needed.
This is what causes rigor mortis after death. The
ATP is needed to break the links between the actin and myosin so allowing the
sliding filament cycle of muscle contraction to be repeated. This occurs late after death therefore the
body relaxes. |
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Q10.
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Breast
meat of turkey is light in colour as it is fast twitch muscle. They contain relatively little
myoglobin. They allow fast muscle
contraction. Predators possess many
fast fibres for fast reactions to capture prey. Leg
meat is dark as it is slow twitch muscle, which is suited to long-term slow
contractions. They contain much more
myoglobin. They consume less fuel
than fast muscle. |